The genomic character of MM is the chromosome translocations via juxtaposition of a set of genes to the immunoglobulin heavy chain locus, which results in overexpression of the translocalized genes such as CCND1, CCND3, MAF, MAFB, MMSET, and FGFR3 [2]. The gene discussed is CCND1; the disease is Miyoshi myopathy.