For rats with endotoxemia and sepsis, a precut bilateral abdominal vagus nerve or injection of α7nAChR blocks α-BGT; the anti-inflammatory and organ protective effects of EA at Zusanli diminished or disappeared, which resulted in the inflammatory factor levels of plasma or tissue inflammatory elevated [4, 9, 11]. This evidence concerns the gene CHRNA7 and serum lipopolysaccharide activity.