TNNT2 and dilated cardiomyopathy: Nonetheless, a chronic adaptive utilization of Frank‐Starling mechanism to compensate for the impaired systolic function would increase energy expenditure and induce dilatative ventricular remodeling, leading to the progression of dilated cardiomyopathy in cTnT‐ΔΕ7 and ΔΕ7+eTnT hearts (Yu et al. 2012).