It has also been suggested that an altered ratio between amylin andinsulin secretion encourages abnormal feeding behaviour under some conditions, such as type1 diabetes or later stages of type 2 diabetes mellitus(16); if such a mechanism were relevant in overweight cats, the discrepancyobserved between amylin and insulin secretion may be one of the factors involved in thedysregulation of satiation and in BW increase in overweight cats. This evidence concerns the gene INS and diabetes mellitus.