VEGFA and posterior cortical atrophy: There are also many reports that SFKs are abnormally activated in PCa cells (1–3), in response to numerous and interconnected stimuli including neuroendocrine ligands (26–28), reactive oxygen species (29), cytokines such as Il-8 (30, 31), growth factors like EGF (27, 29, 32), IGF-1 (28, 33) VEGF (34, 35), or even intracellular activating proteins such as FAK (36).