Platelets sustain the inflammatory process at all stages of atherosclerosis by expressing membrane molecules such as intercellular adhesion molecule-2 (ICAM-2), P-selectin, CD95L, and CD40L that regulate several biological functions in the vessel wall, including cellular adhesion and aggregation, chemotaxis, survival and differentiation, and angiogenesis [3, 4]. Here, ICAM2 is linked to atherosclerosis.