Figure 6(b) shows the results of NF-κB activation in whole lungs from the five groups of mice using an electrophoretic mobility shift assay (EMSA). NF-κB-specific DNA-protein binding activity was markedly increased in lung tissues in the Der p group. In contrast, mice treated with FUT, FOY, and UTI showed markedly inhibited NF-κB activity compared with those treated with Der p. The gene discussed is NFKB1; the disease is bacterial urinary tract infection.