The neurobiology of cognitive dysfunction in schizophrenia is complex, and has been associated with aberrant diffusion and connectivity in the corpus callosum (19–21), dysfunctional oscillations (70, 71) possibly linked to GABA (γ-aminobutyric acid), parvalbumin interneurons, NMDA (N-methyl-d-aspartate) receptors, and cortico-cortical connections (72, 73). The gene discussed is PVALB; the disease is schizophrenia.