In contrast, we saw few CD83+ or CD206+ DCs in the lung in vivo at remission after treatment with corticosteroids, immunosuppressants, and IVIG, but this therapy did not affect the number of eosinophils, which is already high in patients with EGPA before the onset of vasculitis. The gene discussed is CD83; the disease is eosinophilic granulomatosis with polyangiitis.