[33] The mechanism of action may involve the maintenance of oncotic pressure and scavenging of toxic substances present in blood, which reduces the risk of intracellular edema and neurological injury. [34] In addition, lower albumin levels may occur in cases of malnutrition, which has been linked to decreased DTI indices in liver cirrhosis. [35] Our findings suggest that microstructural changes that result from cerebral edema might arise from decreases in albumin levels, and may reverse during clinical recovery. This evidence concerns the gene ALB and malnutrition.