[33] The mechanism of action may involve the maintenance of oncotic pressure and scavenging of toxic substances present in blood, which reduces the risk of intracellular edema and neurological injury. [34] In addition, lower albumin levels may occur in cases of malnutrition, which has been linked to decreased DTI indices in liver cirrhosis. [35] Our findings suggest that microstructural changes that result from cerebral edema might arise from decreases in albumin levels, and may reverse during clinical recovery. The gene discussed is ALB; the disease is nutritional deficiency disease.