The fact that a number of genes differentially regulated in Ity3, Ity3.RecN, and Ity3. RecG strains, were co-expressed with Ncf2, supports the important contribution of the Ity3.1 locus on the pathogenesis of infection in MOLF/Ei and its interaction with the other sub-locus Ity3.2 to enhance the impact of Ncf2 on ROS production. This evidence concerns the gene NCF2 and infection.