As evidence that autoimmune mechanisms, acting via neuroimmunoendocrinological pathways, could be involved in eating disorders, Fetissov et al. [9] reported that a significant subset of patients with AN and bulimia nervosa (BN) had autoantibodies against α-melanocyte-stimulating hormone (α-MSH) and against adrenocorticotropic hormone (ACTH), which contribute to food intake and body weight. The gene discussed is POMC; the disease is eating disorder.