Immunohistochemistry-based morphoproteomic analyses of the tumor specimens from 2 patients (8 and 10) revealed evidence of MAPK pathway activation via p-ERK1/2 and mTOR pathway activation via p-mTOR, as well as heat shock protein 90 (HSP90), tumor necrosis factor–related apoptosis-inducing ligand (TRAIL), and nestin expression. The gene discussed is MTOR; the disease is neoplasm.