TOR1A and Dystonia: Admittedly, this phenotype differs from the pronounced generalized dystonia known from patients suffering from DYT-TOR1A, however, it must be kept in mind that—as we learned from other transgenic models of neurodegenerative diseases (like the transgenic models of Huntington’s disease (Figiel et al., 2012))—motor abnormalities in rodents often differ considerably from those seen in humans.