The expression of this protein is rapidly induced in response to renal tubular injury, and increased levels of serum and urinary NGAL (uNGAL) have been reported in the setting of different renal diseases such as acute kidney injury (AKI), diabetic nephropathy, nephritic syndrome, tubulointerstitial damage, and IgA nephropathy [40–43]. Here, LCN2 is linked to acute kidney injury.