We tested the hypothesis that GRP is linked to the pathogenesis of BPD through analysis of two baboon models of BPD: hyperoxia (140-day-old animals [∼32 weeks human gestational equivalent] given 100% O2 for 10 days, vs. non-BPD 140-day-old animals given PRN O2) and barotrauma (125-day-old animals [∼26 weeks human gestational equivalent] given PRN O2 for 14 days) in collaboration with Jackie Coalson and the NIH Program in BPD (70–72). Here, GRP is linked to bronchopulmonary dysplasia.