The potential role of infection in the pathogenesis of VKH disease is supported by the presence of Epstein-Barr virus DNA in vitreous aspiration of a patient with VKH disease [[16]], and cross-reactions between melanocyte peptides and cytomegalovirus envelope glycoprotein resulting in melanocyte proliferation [[17]]. The gene discussed is ERVW-1; the disease is Vogt-Koyanagi-Harada disease.