Besides, many N-glycosyalted molecules which may also be involved in other types of cancer were also detected in our results, e.g. elevated ALCAM shedding in colorectal cancer was correlated with poor patient outcome [44], BMPR2 participated in the regulation of endoglin-mediated suppression of prostate cancer invasion [45], CD36 was demonstrated to be an important index for predicting the occurrence and development of radiation pneumonitis and evaluating the curative effect [46], etc. Here, ALCAM is linked to Familial prostate cancer.