Bid et al. suggest that ΔNp63α contributes to the progression of OS and neuroblastoma by inducing tumor angiogenesis, because they found that lung sections from patients with OS express high levels of ΔNp63α compared with the primary lesions from the same patients and that this promotes secretion of IL-6 and IL-8 to induce angiogenesis. The gene discussed is IL6; the disease is neuroblastoma.