Given the significant effect of ZNF367 on cellular adhesion, migration, and invasion, which are the necessary steps for cancer progression, we investigated the possible gene(s) that may inhibit this progression as a result of high levels of ZNF367 in cancer, and we found that ITGA3 expression is modulated by ZNF367 and miR-195, and that it is inversely correlated in human tumor samples with ZNF367 expression. Here, ZNF367 is linked to neoplasm.