Because Clec9aΔSyk mice were used as homozygotes in these experiments and therefore lacked DNGR-1 expression [44], we confirmed that DNGR-1 deficiency does not impact on susceptibility to candidiasis by assessing fungal burden in infected Clec9aegfp/egfp mice [45] (Figure 1D). Here, CLEC9A is linked to candidiasis.