We have been able to reproduce this finding (unpublished observations) and therefore, at present, we are forced to conclude that the key mediator of resistance is either a novel IL-23p19-containing cytokine (including, possibly, an IL-23p19 homodimer) or that IL-23 deficiency impairs resistance to candidiasis in an IL-12-sufficient but not IL-12-deficient background. The gene discussed is IL23A; the disease is Candidiasis, Invasive.