Khanna et al., in their bioinformatics analysis of overexpression of CIP2A and components of the EGFR-MEK1/2-ETS1 pathway from two different genome wide leukemia studies have identified M6 subtype of acute myeloid leukemia as a cancer type in which CIP2A and representative genes of each level of the pathway (EGFR, MEK2 and ETS1) were significantly upregulated. This evidence concerns the gene CIP2A and cancer.