Normal human bone marrow was used as a control.6 The lowest level of HMGA2 expression was detected in normal human bone marrow, but the difference between normal human bone marrow and the T-ALL subsets was not substantial, indicating that either HMGA2 did not have an essential role in the development of the cohort of T-ALL cases analyzed6 or that the level of HMGA2 transcripts did not reflect the actual level of HMGA2 protein. The gene discussed is HMGA2; the disease is acute lymphoblastic leukemia.