Since ZIP13 protein forms a homo-dimer (Bin et al, 2011) and the G87R mutation in the zinc transporter ZnT2 is reported to cause neonatal zinc deficiency due to a dominant-negative effect on its homo-dimerization (Lasry et al, 2012), we next examined whether the G64D mutation affects the oligomeric state of the ZIP13 protein. The gene discussed is SLC30A2; the disease is Zinc deficiency.