TRIAP1 and prostate adenocarcinoma: We have shown that miR-203 influenced the mRNA stability of candidate miR-203 targets that are either anti-apoptotic proteins (e.g. API5, BIRC2, and TRIAP1) or the novel oncogenic molecule: TNFAIP8. TNFAIP8 is an NF-κB-inducible molecule that has been shown to be overexpressed in high-grade prostatic adenocarcinomas [45].