Agents targeting molecules such as epidermal growth factor receptor (EGFR), vascular endothelial growth factor (VEGF) receptor, and fibroblast growth factor receptor (FGFR), as well as pathways such as c-Met pathways, are promising candidates for targeted therapy for gastric carcinoma and are now in clinical development [3,4]. This evidence concerns the gene EGFR and gastric carcinoma.