A 5 bp (CTTCT) polymorphism (Fig. 1M;a-c) that results in skipping of exon 4 of TNNT2 during splicing was not significant, when compared to normal controls, it was found to be almost equal in DCM however the deletion frequency was high in HCM [25]. The gene discussed is TNNT2; the disease is familial dilated cardiomyopathy.