Among the roles of mirSNPs in pathogenesis of cardiovascular diseases (CVDs), the mirSNPs rs5186*C (A/C) in human angiotensin II type 1 receptor (AGTR1) 3′-UTR affecting miR-155 binding site [35, 36] and rs9818870 (C/T) in muscle RAS oncogene homolog (MRAS) 3′-UTR modulating miR-195 and miR-135 were proposed to be potentially involved in the hypertension (HT) and other related CVDs [37]. Here, MRAS is linked to hematocrit.