The finding of IL-15, MCP-1 expressions in the cardiac specimens of early infarction confirm a considerable amount of existing data that show the role of humoral (cytokines and inducible chemokines, complement, and toll-like receptors) and cellular (monocytes, macrophages, dendritic cells, T cells, mast cells, platelets, endothelial cells) mediators in the initial healing phases following cardiomyocytes death [4,6,17,24-31]. Here, CCL2 is linked to infarction.