FAF1 and Miyoshi myopathy: In addition, deletions of genes involved in regulation of the NF-κB pathway (CYLD, TRAF3, BIRC2, and BIRC3), cell cycle (CDKN2C, CDKN2A, and CDKN2B), or induction of apoptosis (WWOX and FAF1) were furthermore described by genome-wide approaches and add important information about genetic changes in MM pathogenesis [8, 9].