KDR and neoplasm: While our results establish that the VEGFR2-specific inhibitor DC101 suppresses tumor regression and the recruitment of innate immune cells (Figure 2), our findings do not require that VEGFR2 necessarily be expressed on the tumor-infiltrating innate immune cells themselves, or if it is, that DC101 inhibition of innate immune cell VEGFR2 signaling be the underlying mechanism for the block in immune cell recruitment.