Although we detected mild increases in AAA incidence in all mice that received donor CD8+ T cells from Apoe−/− mice or Apoe−/−Fcer1a−/− mice, and there were complete reverses of the post-Ang-II mortality rate in mice that received CD8+ T cells from Apoe−/− mice but not from Apoe−/−Fcer1a−/− mice, suprarenal aortic diameters from Apoe−/−Fcer1a−/− mice that received donor cells from Apoe−/− mice or Apoe−/−Fcer1a−/− mice were not different from those of the parental Apoe−/−Fcer1a−/− mice (Supplementary Fig S9C). This evidence concerns the gene AGT and triple-A syndrome.