They supposed two progression pathways of IPNB to tubular adenocarcinoma and mucinous carcinoma, featuring the phenotypes of MUC1+/MUC2+ and MUC1−/MUC2+, respectively, which are analogous to that of IPMN of the pancreas. Here, MUC1 is linked to pancreatic intraductal papillary-mucinous neoplasm.