A significant amplification occurred at 1pter and encompassed NOCL2, PLEKHN1, SDF4, UBE2J2, CENTB5, CPSF3L, MXRA8, ATAD3B, ATAD3A, SSU7A, SLC35E2, NADK, GNB1, GABRD, and PRKCZ. A focal amplicon at 12p contains KRAS, which is also mutated in this patient’s tumor. The gene discussed is ATAD3A; the disease is neoplasm.