Since HR-HPV infection was an indispensable factor for cervical cancer and precancerous lesions, and numerous studies have demonstrated that HPV may target a group of molecules, including mTOR, miR-29 and MAGI-1 (33–35), it was hypothesized that HPV may be a key factor inducing MALAT1 expression in cervical SCC. This evidence concerns the gene MALAT1 and cervical carcinoma.