The levels of FCP and 2 h PCP in the patients with ketosis-onset diabetes was intermediate between those with type 1 and type 2 diabetes, partly explained by the fact that ketosis-onset diabetes has a predominant insulin secretory defect rather than a decline in beta-cell mass months after recovery from the index episode of ketoacidosis[4,34,35]. Here, INS is linked to type 2 diabetes mellitus.