In contrast to previous observations in mice, this study reported a lack of CCL19 or CCL21 protein expression in endothelial or parenchymal cells of non-lesioned white matter or in active or chronic MS lesions, but did find strong CCL21 immunoreactivity within the choroid plexus epithelium (Kivisäkk et al., 2004). This evidence concerns the gene CCL21 and myeloid sarcoma.