Thus, if indole is present at a sufficient concentration in the infection microenvironment, genital serovars could circumvent the bactericidal/bacteriostatic effects of IFNγ (Beatty et al., 1993; Fehlner-Gardiner et al., 2002; Belland et al., 2003; Caldwell et al., 2003). Here, IFNG is linked to infection.