In [32, 33], a model for glioblastoma growth was introduced that combines an agent based model with an EGFR (epidermal growth factor receptor) signalling network and focuses on the determination of the cell phenotypes “migrating” and “proliferating.” Later, also a model for the progression of lung cancer was developed [34] in that essentially only the molecular interaction network was interchanged. The gene discussed is EGFR; the disease is lung carcinoma.