We identified SNPs near TNFRSF9 at which psoriasis susceptibility variants disrupted binding sites for AP-1 (rs6687168) and NF-κB (rs6661746), where both binding sites were also enriched with respect to sequences adjacent to genes co-expressed with TNFRSF9 in neutrophils (Figures 7 and 8). The gene discussed is FOS; the disease is psoriasis.