The involvement of MBL-MASP complexes in haemostatic processes was later demonstrated in an animal model by Takahashi et al. [33] who found that MBL-null mice infected with Staphylococcus aureus were predisposed to the development of disseminated intravascular coagulation (DIC). The gene discussed is MBL2; the disease is Disseminated intravascular coagulation.