Xeroderma pigmentosum (XP) displays several OS features, including defective repair of DNA oxidative damage (8,5′-(S)-cyclo-2′-deoxyadenosine), while lipid peroxidation and protein glycation are increased in cells and brains from XP patients, along with reduced expression of SOD [73]. Here, SOD1 is linked to xeroderma pigmentosum.