The lack of correlation in our control rats suggests that the surgery itself did not cause the behavioral impairment, and that the deficit seen in KIA- rats was due to RNAi of the candidate-dyslexia gene Kiaa0319. Our results suggest that the degree of in utero transfection of this gene is related to each rats' aptitude for learning the full speech sound task. The gene discussed is KIAA0319; the disease is dyslexia.