IAV infection significantly increased the levels of all tested proinflammatory cytokines (IL-1β, IL-6, IL-2, TNF-α, IFN-α, IFN-β and IFN-γ) in lung homogenates between 1.2- and 13.7-fold relative to the baseline (before infection) (Figure 5). This evidence concerns the gene IFNB1 and infection.