Furthermore, deletion of inlB in the actA deletion strain that expressed the murine colon TAA, gp70, resulted in increased immunogenicity in comparison to actA deficiency alone as observed by significantly greater gp70-specific CTL responses, reduced metastatic burden with CT26 colon carcinoma tumors, and increased survival (Olino et al., 2012). The gene discussed is EMB; the disease is colon carcinoma.