The results indicated that pim1 was also weakly oncogenic in the prostate environment, but it cooperated dramatically with c-myc to induce prostate cancer within 42 days, generating large hemorrhagic tumors, while the control, pim1, K67M, or c-myc or c-myc/K67M grafts were small and did not differ significantly in their sizes. The gene discussed is MYC; the disease is prostate carcinoma.