There were no significant differences in age, gender distribution, cardiac output, free T4, free T3, TSH, creatine phosphokinase, CRP levels, cholesterol, serum albumin, white blood cells or incidence of medical therapy (beta-blocker and/or thiamazole) between patients with and without pulmonary hypertension (Tables 1, 2). Here, CRP is linked to pulmonary arterial hypertension.