LPL and lipodystrophy: As corticoids are well known to depress the inflammatory response [38] and to induce lipodystrophy through the decreased expression of lipoprotein lipase, an enzyme secreted by adipocytes and involved in triglyceride synthesis [39], we hypothesize that the defective immune response and the lipodystrophy induced under corticotherapy may favor the release of pathogens from AT and, consequently, the reactivation of infection.