As expected based on our data, complete IL-17R−/− BMC mice (−/−host/−/− BM) were more susceptible to Mtb HN878 infection than complete B6 BMC mice (B6 host/B6 BM) (Fig. 7A), and demonstrated defects in T cell localization (Fig. 7B), formation of lymphoid follicles and CXCL-13 mRNA expression (Fig. 7C), and CXCL-13 protein expression within B cell lymphoid follicles (Fig. 7D). This evidence concerns the gene IL17RA and infection.