Sobngwi et al. had described a high prevalence of glucose-6-phosphate-dehydrogenase (G6PD) deficiency without gene mutation in these patients [27]; then Choukem et al. reported that polymorphism of Arg585Gln in SREBF-1 was not associated with the KPD phenotype [28]. The gene discussed is G6PD; the disease is G6PD deficiency.